| Titles:
Research Associate, Department of Critical Care Medicine
Contact:
Email:hanx@ccm.upmc.edu
Degrees:
POST GRADUATE EDUCATION
INSTITUTION AND LOCATION |
DEGREE |
YEAR |
FIELD |
| Peking Union Medical College (PUMC) & Chinese Academy
Medical Sciences (CAM) |
|
1995 |
|
Other:
|
|
| Peking Union Medical College (PUMC) & Chinese Academy
Medical Sciences (CAM) |
Ph.D. |
1995-2000 |
Pathology |
Research Grants:
| Title |
Source of Support |
| Molecular Mechanisms of Gut Barrier Dysfunction |
NIH-GM58484-02 |
Current Research Interest:
- Tight Junction of Epithelial cells and Multi-organ
Dysfunction
Publications:
- Han X, Fink, MP, Delude, RL. Proinflammatory
Cytokines Cause NO·-Dependent and Independent Changes in
Expression and Localization of Tight Junction Proteins in Intestinal
Epithelial Cells. Shock, 2003, 19(3):229-237.
- Han X, Sappington, PL, Uchiyama, T et al.
Extracellular NAD Preserves Mucosal Barrier Dysfunction in Immunostimulated
Caco-2 Cells and Endotoxemic Mice, Gastroenterology, submitted
as rapid communication.
- Han X, Yang, R, Fink, MP et al. LPS-Induced
Structural and Functional Alterations in Tight Junctions in Multiple
Organs of Mice is Nitric Oxide-Dependent. submitted.
Presentations at Major Meetings:
- Han X, Yang, R, Fink, MP, Delude, RL. Lipopolysaccharide
(LPS) Administration to Mice Induces Dysregulation of Tight Junction
Formation in Multiple Organs Through a NO·-Dependent Mechanism.
Molecular Cell Biology, 2002,13:271a (42nd ASCB Annual Meeting,
2002, San Francisco, CA)
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